tissue. Survival for more than 48 hours usually
indicates that recovery is possible.

Bariun carbonate is the usual source of barium in cerami
usage. It, along with barium chloride and barium
hydroxide, is partially soluble and may be a deadly
poison. If completely absorbed into the system, as
little as one gram can be fatal. The first symptoms
are tremors followed by convulsions and, in terminal
cases, death due to cardiac and/or respiratory failures.

Inhaled barium compounds are also dangerous, but are
not usually absorbed into the system and, hence, not
fatal. They produce "Baritosis," a form of pneumoconios
associated with barite.

Beryllium compounds can be taken into the body by
ingestion, inhalation, or through cuts. Cuts coming
in contact witn beryllium compounds may become ulcer-
ated and require months to heal. Beryl]lium oxide is a
strong sensitizer, and the dust may cause dermatitis
similar to first and second degree burns.

About one-quarter of all beryllium taken into the
system is deposited in the bones and becomes permanent.

Inhaled dust is by far the most dangerous. It causes

a granulation of the Jungs and symptoms similar to
silicosis. A single massive dose could cause death
almost immediately, or the reaction may be delayed for
weeks. Small amounts taken in over a period of time can
produce pneumonia or allergic sensitivities. Again,

the reaction may be delayed for up to fifteen years.

Borie actd, borax and sodiwn perborate are all poisons
according to Moeschlin. Fatal doses taken internally
vary from two grams for infants to twenty grams for
adults, but individual tolerances vary widely. A
fatal dose could be absorbed through a cut or skin
abrasion. An acute dose leads to vomiting, diarrhea,
and sometimes collapse and hemorrhaging.

Chronic poisoning can cause a variety of symptoms,

including anemia, delusions, diarrhea, and skin
disorders similar to psoriasis,

3]